Orthoses
Clubfoot and Ponseti: joint effects and the role of bracing
Ponseti corrects over 90% of clubfeet without major surgery, yet the ankle stays weaker and relapse is common. An evidence summary on the ankle, knee, hip, Achilles tendon and which braces help avoid surgery.
Ponseti is the best treatment for clubfoot in infants today, but after treatment the ankle remains weaker than in typically developing children, and the knee and hip have to “work extra” to compensate when walking. Clubfoot also tends to relapse, so the bracing phase is crucial.
This article summarises studies from PubMed, PMC and Cochrane in plain language: how Ponseti affects the ankle, knee and hip; whether casting and bracing cause harm; why the Achilles tendon tends to tighten again; and which orthoses help reduce relapse and avoid surgery.
Quick summary
- Ankle (strong evidence, meta-analyses): after Ponseti, children push off more weakly, have a narrower ankle range of motion, and the foot tends to point inwards when walking.
- Knee and hip: the changes are mainly compensatory. The knee straightens slightly less, and the hip rotates outwards so that the foot does not point inwards.
- Casting and bracing: the recorded complications involve the skin and the back of the knee. No large study has shown that casts or braces damage the knee or hip joint.
- Achilles tendon: if the brace does not hold the foot lifted up (dorsiflexion), the Achilles tendon easily tightens again. This is the most common type of relapse; a second Achilles tenotomy is the most common additional operation.
- Late stage (relapse, late treatment): in gait-analysis studies, 50–70% of relapsed children rotate the hip outwards to compensate, 71% have internal tibial torsion and 29% have knee hyperextension; about 30% of relapsed children had needed a second tenotomy.
- A conventional ankle-foot orthosis (AFO) that only holds the foot straight is not enough: in one comparative study, 83% relapsed with an AFO versus 31% with a Denis Browne brace.
Clubfoot and the Ponseti method
Clubfoot is a congenital deformity: the foot is twisted inwards, the heel is pulled up and turned in, and the toes point down. Without treatment the child walks on the outer edge or the top of the foot, causing pain and disability.
The Ponseti method has 3 phases:
- Manipulation and casting: each week the foot is gently corrected and cast from the toes to the groin with the knee bent at 90°. Usually 5–7 casts are needed.
- Percutaneous Achilles tenotomy: most children need a small cut in the Achilles tendon so the foot can be lifted up. A further cast for about 3 weeks lets the tendon heal at its new length.
- Foot abduction brace (Denis Browne): two shoes joined by a bar. The affected foot is turned out 60–70° and lifted 10–15°. It is worn almost all day for the first 3 months, then during sleep until age 4–5.
Ponseti corrects over 90% of children without major surgery and is regarded as today’s gold standard. However, clubfoot tends to relapse, so the bracing phase is crucial.
How to read the evidence: levels of confidence
Not every study carries the same weight. This article grades the evidence in 3 levels so readers know what is certain and what is still a hypothesis.
| Level | Type of study | What it means for the reader |
|---|---|---|
| Strong | Systematic reviews, meta-analyses (pooling many studies), Cochrane reviews | Highly reliable |
| Moderate | Cohort studies, comparisons with a control group | Fairly reliable but needs confirmation |
| Weak | Single cases, small groups under 30 children, expert opinion | Suggestive, not enough to conclude |
Sources were searched in PubMed (Medline), PMC and the Cochrane Library. The systematic reviews cited also searched Embase, Web of Science and Scopus.
After treatment: gait at the ankle, knee and hip
A weak ankle and an in-pointing foot are the root of the problem; the knee and hip change to compensate. The results below come from 3D gait analysis in the laboratory (cameras and force plates).
Ankle – strong evidence
- Meta-analysis of 20 studies (Grin et al., 2023): compared with healthy children, children after Ponseti have less plantarflexion at push-off, less dorsiflexion in swing and a significantly narrower ankle range of motion. The foot also points inwards more when walking.
- Meta-analysis of kinetics (Tuinsma et al., 2018): ankle push-off moment and power are lower than in healthy children. However, they are no worse than in children treated surgically.
Put simply: the ankle is a weaker “spring” and pushes the body forward less well.
Knee – moderate evidence
- In mid-stance, the knee of children after Ponseti extends less fully than in healthy children.
- When the foot points inwards, the ground reaction force shifts behind the knee. One study recorded about 7° more knee flexion and about 60% higher knee flexion moment in stance (in treated clubfoot in general, not Ponseti only).
- Children treated with French physiotherapy often walk with knee hyperextension (41% of feet), whereas children treated with Ponseti casting do not toe-walk.
- After the old extensive release surgery, the knee often shows valgus, mild hyperextension and quadriceps weakness. This is why Ponseti is preferred to surgery.
Hip – moderate evidence
- Follow-up to age 5 (Karol et al., 2009): 85% of Ponseti children had in-toeing of the leg and foot, and 71% of limbs had external hip rotation in stance to compensate.
- When ankle push-off is weak, the knee and hip must generate more power to compensate. The authors call this a compensatory mechanism.
- Clubfoot itself is associated with increased internal hip rotation, so not every hip change is caused by the treatment.
The compensation chain: weak ankle push-off and an in-pointing foot → more knee flexion and load → the hip rotates outwards and adds power to keep the step straight.
During casting
Casts must run from the toes to the groin with the knee bent at 90°. The effect on the knee that has been recorded is mainly pressure behind the knee, not joint damage.
Why cast above the knee? With a below-knee cast the leg can rotate inside the cast and the cast slips. One study recorded slippage in 37.5% of below-knee casts versus 5.9% of above-knee casts. Another randomised trial was stopped early because below-knee casts failed so often.
Recorded side effects:
| Side effect | Frequency / severity | Level of evidence |
|---|---|---|
| Skin injury (redness, peeling, sores) | About 13% of casting episodes; about 20% of children | Moderate |
| Pressure or sores behind the knee | More likely when the knee is flexed beyond 110° to stop slipping | Weak (expert opinion) |
| Loose or slipped cast | 5.5% of feet in a case series from Thailand | Moderate |
| Osteopenia, muscle wasting from immobilisation | With prolonged casting | Weak |
| Deformity from incorrect manipulation (cavus, heel valgus) | Caused by technique, not by the method | Moderate |
The 2020 Cochrane review noted that most trials did not report side effects; those that did recorded only cast slippage, cast sores and skin irritation. No report described knee contracture, knee ligament laxity or hip damage from casting.
During Denis Browne bracing
The brace deliberately transmits force to the leg, thigh and hip; current studies have not found this force to deform bones or joints, but they are small.
How does the brace work? Both feet are fixed to a bar. The external rotation force travels up through the tibia and femur, preventing the knee and hip from turning inwards to “escape” the corrective force. This is intentional and part of the treatment.
What has been measured:
- Bone torsion: a study of 20 children who wore the brace for an average of 33 months, measured by ultrasound. Femoral anteversion and external tibial torsion stayed within normal limits; the authors concluded the brace caused no pathological change (weak evidence because the group was small).
- Flattening of the healthy foot: in unilateral clubfoot the healthy foot also wears the brace and becomes clearly flatter during this period (moderate evidence).
- Skin: about 14% of children using the Mitchell–Ponseti brace had skin abrasions, mainly on the top of the foot.
Unproven concerns: some experts warn that excessive external rotation (70°) might cause excessive external tibial torsion (out-toeing) and genu valgum (knock knees). This is expert opinion, not confirmed by measurement studies. The University of Iowa (where Ponseti worked) advises that the bar should be as wide as the child’s shoulders for comfort and to avoid knee and hip problems.
About the hip: a 2015 meta-analysis found the rate of hip dysplasia in clubfoot similar to that in other children. Other studies, however, report about 1 in 17 children with clubfoot having hip dysplasia, so many centres still screen the hips by ultrasound. In children with both hip dysplasia and clubfoot treated at the same time with a Pavlik harness and Ponseti, a small case series recorded abnormal femoral neck growth (coxa valga) and 1 case of femoral head avascular necrosis; it was not possible to separate the effect of the brace from that of the harness (weak evidence).
The Achilles tendon, relapse and repeat tenotomy
If the brace does not hold the foot lifted up, the Achilles tendon easily tightens again; this is the most common type of relapse and often needs a second tenotomy.
The mechanism is simple: after the cut, the Achilles tendon heals at a new length. But a clubfoot tends to “pull back” towards its old position. The brace has 10–15° of built-in dorsiflexion to stretch the calf and tendon continuously. If the heel rides up out of the shoe, or the child does not wear the brace long enough, the tendon loses that stretch and gradually shortens. The foot then points down again, known as recurrent equinus.
| Finding | Figure | What the figure means |
|---|---|---|
| Non-adherence to bracing is the main cause of relapse | 86% | Share of relapsed feet with non-adherence (of 115 relapsed feet) |
| Most common component of relapse | — | Equinus, followed by forefoot adduction |
| Non-adherence increases the risk of relapse | 5 to 183 times | Increase in risk, varying between studies |
| Most common additional operation after Ponseti | 41.2% | Repeat tenotomy as a share of all additional operations |
| Repeat tenotomy in a large US cohort | 5.75% | Share of all treated children |
| Achilles tendon after the first tenotomy | — | Fully healed when re-operated |
Early warning sign: if, after taking the brace off, the heel has not touched the sole, or the foot keeps slipping out of the shoe, consider early relapse. A foot that does not lift enough slips out of the brace more easily, creating a vicious circle.
Dynamic bars: a hinged, spring-loaded bar (such as the Dobbs bar) is more comfortable. But the child can overpower the spring and point the foot down at times, and the calf and tibialis posterior muscles may strengthen and contribute to relapse. This is only a statement in a study protocol; there are no results yet.
Late stage: relapse, late treatment and repeated tenotomy
When clubfoot relapses or is treated late, the consequences are not limited to the foot: the leg twists inwards, the hip turns outwards to compensate, the knee loads differently, and the Achilles tendon may need cutting several times.
“Late stage” here includes children treated with Ponseti who relapsed, children not fully corrected, and children first seen after walking age (over 1 year), even at 8–10 years.
- The probability of relapse can reach 52% by age 6 and falls clearly with correct bracing; with correct bracing, only about 12% need surgery.
- In Ponseti’s own study: 7% relapsed among children who braced correctly versus 78% among those who did not.
- Children treated after walking age: a meta-analysis of 654 feet found a relapse rate of 18%.
- A multicentre study of 429 feet achieved correction in 87%, but 31% relapsed, mainly because of non-adherence to bracing. Among children aged 1–10, about 31% relapsed and needed re-casting, re-bracing or tendon transfer.
Effects on the ankle, knee and hip in relapse
Gait analysis in relapsed children shows a clear chain of compensation from the bottom up (figures are the share of children or feet with that sign in each study):
| Site | Change in relapse | Frequency |
|---|---|---|
| Foot | In-toeing gait | 73–80% |
| Foot | Supination in stance from an overactive tibialis anterior | 70% of supinated feet |
| Ankle | Reduced push-off (plantarflexor moment) | 83–84% |
| Leg | Internal tibial torsion | 71% |
| Knee | Knee hyperextension when walking | 29% |
| Knee | Increased knee flexion moment (unilateral clubfoot) | Recorded, no rate given |
| Hip | External rotation to compensate | 50–70% |
In plain words: when the foot and leg are twisted inwards, the child would trip if they kept walking that way. The body “fixes” it by turning the whole thigh outwards at the hip. The foot looks straighter, but the hip is working against its natural direction. This compensatory hip rotation is often invisible to the eye, is detected only by gait analysis, and can mask the true degree of deformity in the leg and foot.
Late relapse (after age 4): more external hip rotation, more internal leg rotation and weaker ankle push-off; compensations at the hip, knee and foot are always present. A 2023 meta-analysis: relapsed children have less external knee rotation and more external hip rotation in stance; in swing, more knee flexion and more hip abduction.
The Achilles tendon and repeated tenotomy
- Among relapsed children, about 30% had already needed a second tenotomy. Both the clubfoot group and the relapse group had shorter calf muscles, longer Achilles tendons and poorer walking function than typically developing children; the authors believe tenotomy is not the only cause.
- The calf on the clubfoot side is smaller than the healthy side from the start. The first tenotomy does not worsen calf atrophy in the short term.
- The Achilles tendon can heal completely after tenotomy, even in older children (average age 5), and calf strength returns to normal.
- Children under 2 who relapse usually have another percutaneous tenotomy. Older children usually need an open Z-lengthening of the Achilles tendon. Recurrent equinus is the most common reason for re-operation after Ponseti.
Why try to avoid surgery at the late stage? Children over 8–10 who relapse often need bone cuts and corrections, sometimes with an external fixator. The last resort is fusion of three joints of the foot; long-term results in children are lacking and the prognosis is considered poor. Children who have major surgery often develop stiffness, pain and early arthritis in adulthood.
The role of orthoses at the late stage
At the late stage, the orthosis is the last line of defence before surgery: the right brace, worn long enough, clearly reduces relapse and surgery. But not every brace is equally effective.
The sequence supported by the literature: re-casting with Ponseti (even in older children) → tenotomy if needed → maintenance orthosis → surgery only if the brace cannot hold the correction. Bone surgery is reserved for older children, usually over 10.
| Finding | Figure | Level of evidence |
|---|---|---|
| Longer bracing reduces surgery | 279 feet followed for 6.3 years; only 12.9% needed further surgery | Moderate |
| Duration of bracing | 2 years of bracing: 56% relapsed; longer: 11% relapsed (Ponseti’s data) | Moderate |
| Adherence to bracing | Non-adherent: 32–88% relapsed; adherent: 4–22% relapsed | Strong (many studies) |
| After re-casting for relapse | The author advises a moulded ankle-foot orthosis for about 6 months | Weak (expert opinion) |
| Children over 5–6 who cannot tolerate a bar brace | An ankle-foot orthosis may be a useful option (Ponseti International Association guideline) | Professional guideline |
Which braces work?
A conventional ankle-foot orthosis (AFO) that only holds the foot straight is not enough. In a comparative study (Janicki, 2011), 83% of the AFO group relapsed versus 31% of the Denis Browne group. The reason: an AFO only holds the foot in a neutral position and cannot provide the 60–70° of external rotation needed to stretch the soft tissues on the inside of the foot.
An effective brace needs 3 elements (drawn from comparing successful and unsuccessful braces):
- It actively pushes the foot into abduction (outward rotation) and dorsiflexion, not merely “holding” the position.
- It has a support above the knee, with the knee flexed, to correct tibial torsion. A brace that stops below the knee cannot do this.
- The child accepts wearing it for enough hours.
Braces with good evidence
- Custom-moulded dynamic knee-ankle-foot orthosis (dynamic KAFO): 20 children (30 feet) had no relapse during 4 years of bracing, with high adherence. Notably, in-toeing in these children was not compensated by external rotation at the knee or hip, unlike what is often seen after a bar brace.
- Dynamic torsional KAFO (DTKAFO): 88% of children adherent, only 8% relapse after 2 years, significant improvement in ankle range of motion, comparable to a bar brace.
- Custom unilateral lower-leg orthosis: switching from a bar brace to this orthosis at any time did not increase the rate of surgery.
- AFO by day plus a bar brace at night: better ankle range of motion, better function and fewer relapses than a bar brace alone.
- Plantar pressure-distributing orthoses: for relapsed children of walking age, the authors suggest this type may improve foot and ankle function (still a hypothesis).
Practical implications
- At the late stage, the brace must be designed and moulded for each child, based on the remaining deformity (equinus, adduction, varus, tibial torsion).
- The brace must hold the ankle in true dorsiflexion without the heel riding up, because this is the key to preventing recurrent equinus and avoiding another tenotomy.
- When there is tibial torsion or compensatory hip rotation, consider a brace with an above-knee part rather than a below-knee AFO only.
Evidence gap: no large randomised trial has compared brace types in relapsed or late-treated children. Most data come from small cohorts or retrospective studies.
What there is no evidence for yet
So far, no large study has shown that Ponseti casting or bracing causes long-term damage to the knee or hip.
- There is no systematic review, randomised trial or large follow-up study on knee or hip injury from casts or braces.
- No study has followed children into adulthood to see whether the knee or hip develops early arthritis.
- No study has directly measured the “actual dorsiflexion inside the brace” against the rate of repeat tenotomy; studies so far only measure whether parents have the child wear the brace long enough.
- No study has measured knee angle or knock-knee angle in children braced at 70° versus 60° of external rotation.
Absence of evidence is not proof of no harm. Clinical observations of effects on the knee and hip still deserve to be recorded, measured and published to fill this gap.
Practical advice for parents
The most important thing is to wear the brace for the right hours and in the right way until the doctor says to stop, usually at age 4–5.
While your child is in a cast
- Check the toes every day: pink, warm and moving. Toes that are blue, cold, very swollen or retracting into the cast → tell the doctor at once.
- Look at the back of the knee and the cast edges for red skin.
- A cast that is loose, slipping, cracked, soft, wet or smells bad → get it checked; do not wait for the next appointment.
While your child wears the brace
- Wear it almost all day (23 hours) for the first 3 months, then during night sleep and naps.
- Press the heel down onto the sole before fastening; fasten the middle strap (across the ankle) first.
- After taking the brace off, check that the heel has left a mark on the sole. A heel that does not touch the sole, or a foot that keeps slipping out, is an early sign of relapse.
- The bar should be as wide as the child’s shoulders; change the size as the child grows.
- Red marks that have not faded about 1 hour after removing the brace → tell the orthotic clinic. Let the child kick both legs together in the brace to get used to it.
Come back early if you notice the foot is hard to lift, the heel turns inwards, the child walks on the outer edge of the foot, or the knee and foot point outwards too much when walking.
Glossary
| Term | Simple explanation |
|---|---|
| Clubfoot (CTEV) | Congenital deformity; the foot is twisted inwards and points down |
| Dorsiflexion | Lifting the toes up towards the shin |
| Plantarflexion | Pointing the toes down, as when standing on tiptoe |
| Equinus | The foot cannot be lifted to a right angle with the leg |
| Achilles tendon | The large tendon joining the calf to the heel bone |
| Percutaneous tenotomy | A small cut through the skin to lengthen the tendon so the foot can lift |
| Foot abduction brace, Denis Browne brace | Two shoes joined by a bar, holding the feet turned outwards |
| Stance phase | When the foot is on the ground during a step |
| Swing phase | When the foot leaves the ground and moves forward |
| Tibial torsion | How much the shin bone is twisted around its axis |
| Genu valgum | Knock knees; the knees touch |
| Hip dysplasia | Abnormal development of the hip socket; the femoral head dislocates easily |
| Relapse | The deformity returns after correction |
| Meta-analysis | Pooling data from several studies for a stronger conclusion |
If your child is being treated for clubfoot, has relapsed or was diagnosed late, bring the X-rays and records for an examination, gait assessment and advice on a custom orthosis. Hotline +84 985 211 888. This article is for health education and does not replace an in-person examination.
References
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- Tuinsma ABM, Vanwanseele B, van Oorschot L, et al. Gait kinetics in children with clubfeet treated surgically or with the Ponseti method: a meta-analysis. Gait Posture. 2018;66:94–100.
- Karol LA, Jeans K, ElHawary R. Gait analysis after initial nonoperative treatment for clubfeet: intermediate term followup at age 5. Clin Orthop Relat Res. 2009;467(5):1206–1213.
- 3D gait analysis of children treated for idiopathic CTEV after completion of Ponseti treatment.
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- Lower extremity characteristics in recurrent clubfoot: clinical and gait analysis findings. Gait Posture. 2019.
- Gait parameters in children with late clubfoot relapse after initial conservative therapy are different. Gait Posture. 2024.
- Muscle-tendon properties and functional gait outcomes in clubfoot patients with and without a relapse. Gait Posture. 2022.
- Effect of Achilles tenotomy on congenital clubfoot-associated calf-muscle atrophy: an ultrasonographic study. J Orthop Sci.
- Can the Achilles tendon regenerate completely following percutaneous tenotomy in older children with clubfoot? Int Orthop. 2024.
- Masrouha KZ, et al. Narrative review of the management of a relapsed clubfoot. Ann Transl Med. 2021.
- Shabtai L, Segev E, Yavor A, Wientroub S, Hemo Y. Prolonged use of foot abduction brace reduces the rate of surgery in Ponseti-treated idiopathic club feet. J Child Orthop. 2015;9(3):177–182.
- Janicki JA, Wright JG, Weir S, Narayanan UG. A comparison of ankle foot orthoses with foot abduction orthoses to prevent recurrence following correction of idiopathic clubfoot by the Ponseti method. J Bone Joint Surg Br. 2011;93(5):700–704.
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- Farrar EJ, et al. Two-year retrospective cohort results on use of a dynamic unilateral brace (DTKAFO) for treatment of clubfoot. 2022.
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